TruckerGuy:
Franglais:
stu675:
Franglais:
[q?
There is a greater risk of transmitting the virus by unvaccinated persons than vaccinated ones. If infected the virus can be passed on in both cases, but since the unvaccinated are more likely to become infected, they are a greater risk.
.
The opposite is true.
If I’ve got it wrong, please explain.
Antibody Dependent Enhancement.
Just look at the stats. The most vaccinated countries have the highest case rates. It is not a coincidence, and there are more research papers on this phenomenon than I can be bothered to link.
nature.com/articles/s41564-020-00789-5
Data from the study of SARS-CoV and other respiratory viruses suggest that anti-SARS-CoV-2 antibodies could exacerbate COVID-19 through antibody-dependent enhancement (ADE). Previous respiratory syncytial virus and dengue virus vaccine studies revealed human clinical safety risks related to ADE, resulting in failed vaccine trials.
…
ADE has been observed in SARS, MERS and other human respiratory virus infections including RSV and measles, which suggests a real risk of ADE for SARS-CoV-2 vaccines and antibody-based interventions. However, clinical data has not yet fully established a role for ADE in human COVID-19 pathology.
frontiersin.org/articles/10 … 40093/full
Antibody-dependent enhancement (ADE) may be involved in the clinical observation of increased severity of symptoms associated with early high levels of SARS-CoV-2 antibodies in patients. Infants with multisystem inflammatory syndrome in children (MIS-C) associated with COVID-19 may also have ADE caused by maternally acquired SARS-CoV-2 antibodies bound to mast cells. ADE risks associated with SARS-CoV-2 has implications for COVID-19 and MIS-C treatments, B-cell vaccines, SARS-CoV-2 antibody therapy, and convalescent plasma therapy for patients. SARS-CoV-2 antibodies bound to mast cells may be involved in MIS-C and multisystem inflammatory syndrome in adults (MIS-A) following initial COVID-19 infection. SARS-CoV-2 antibodies bound to Fc receptors on macrophages and mast cells may represent two different mechanisms for ADE in patients. These two different ADE risks have possible implications for SARS-CoV-2 B-cell vaccines for subsets of populations based on age, cross-reactive antibodies, variabilities in antibody levels over time, and pregnancy. These models place increased emphasis on the importance of developing safe SARS-CoV-2 T cell vaccines that are not dependent upon antibodies.
medrxiv.org/content/10.1101 … 114v1.full
If you think these people care about your health, think again:
science.org/content/article … ing-safety
The biggest concerns, however, are about the vaccine’s safety. In the largest trial, children who received Mosquirix had a risk of meningitis 10 times higher than those who received a control vaccine. Mosquirix may not have triggered the meningitis cases—there are other possible explanations—but the possible risk worried the global health community so much that, rather than rolling out the vaccine across Africa, the World Health Organization u has decided to set up a pilot in Malawi, Ghana, and Kenya in which the vaccine will be given to hundreds of thousands of children.[/u]
…nothing like doubling-down on a monumental [zb] up, huh!
This is the kicker…from 2012:
pubmed.ncbi.nlm.nih.gov/22536382/
However, challenge of mice given any of the vaccines led to occurrence of Th2-type immunopathology suggesting hypersensitivity to SARS-CoV components was induced. Caution in proceeding to application of a SARS-CoV vaccine in humans is indicated.
It is a fact that the current SARS-CoV-2 “vaccines” cause massive coronary, pulmonary, and other issues in humans. In the past 12 months there has been a 300%+ increase in pro footballers having sudden stroke and heart attack leading to death vs. the 12-year average, and countless hundreds of other athletes (that we know about) have either retired or “taken a break” for sudden heart conditions (again, all following vaccination).
The MSM are normalizing heart conditions in what were perfectly fit and healthy individuals, and for the first time in the medical records, children under the age of 10 are experiencing heart attacks and strokes following vaccination. Of course, the MSM say it is just a “conspiracy theory”.
We have built a time-bomb, and the MSM and Governments are currently doing all they can to bury the truth. To help you feel more at ease, the pharmaceutical industry and the medical staff that administer these “vaccines” have been given immunity from prosecution for any injury or death caused by the “vaccines”.
Like all the best conspiracy theories this starts with a bit of truth and fact to hook you, then tugs you in with exaggeration, false logic, and twists you up.
Ive quoted the whole post above for accuracy and context, but Ill snip out bits to examine:
TruckerGuy:
Franglais wrote:
stu675 wrote:
Franglais wrote:
[q?
There is a greater risk of transmitting the virus by unvaccinated persons than vaccinated ones. If infected the virus can be passed on in both cases, but since the unvaccinated are more likely to become infected, they are a greater risk.
.
The opposite is true.
If I’ve got it wrong, please explain.
Antibody Dependent Enhancement.
Just look at the stats. The most vaccinated countries have the highest case rates. It is not a coincidence, and there are more research papers on this phenomenon than I can be bothered to link.
nature.com/articles/s41564-020-00789-5
Firstly a place with most vaccines has most cases?
Who knows? Countries with best health care will have most vaccines, and will do most tests, so will have most documented cases. If you look at underdeveloped countries the known numbers of cases is low, but the iceberg under the water is unseen. The post doesn`t address case rates from random sampling.
Secondly ADE is a thing and the link does go that article.
“No definitive role for ADE in human coronavirus diseases has been established.”
There is no evidence for it regards Covid.
“Should it occur, ERD caused by human vaccines will first be observed in larger phase II and/or phase III efficacy trials that have sufficient infection events for statistical comparisons between the immunized and placebo control study arms. Safety profiles of COVID-19 vaccines should be closely monitored in real time during human efficacy trials, especially for vaccine modalities that may have a higher theoretical potential to cause immunopathology (such as inactivated whole-virus formulations or viral vectors”
WE need to still check for it.
And
"Conclusion
ADE has been observed in SARS, MERS and other human respiratory virus infections including RSV and measles, which suggests a real risk of ADE for SARS-CoV-2 vaccines and antibody-based interventions. However, clinical data has not yet fully established a role for ADE in human COVID-19 pathology. Steps to reduce the risks of ADE from immunotherapies include the induction or delivery of high doses of potent neutralizing antibodies, rather than lower concentrations of non-neutralizing antibodies that would be more likely to cause ADE.
Going forwards, it will be crucial to evaluate animal and clinical datasets for signs of ADE, and to balance ADE-related safety risks against intervention efficacy if clinical ADE is observed. Ongoing animal and human clinical studies will provide important insights into the mechanisms of ADE in COVID-19. Such evidence is sorely needed to ensure product safety in the large-scale medical interventions that are likely required to reduce the global burden of COVID-19."
So, nothing current but checks need to be ongoing. Sounds OK to me.
And none of this says anything at all about my statement that virus is more likely to be transmitted by non-vaccinated than by vaccinated.
thelancet.com/journals/lani … 73-3099(2100648-4/fulltext
“Interpretation
Vaccination reduces the risk of delta variant infection and accelerates viral clearance. Nonetheless, fully vaccinated individuals with breakthrough infections have peak viral load similar to unvaccinated cases and can efficiently transmit infection in household settings, including to fully vaccinated contacts. Host–virus interactions early in infection may shape the entire viral trajectory.”
Vaccination is not magic. It does reduce risk to the individual and to those around them.
Next as posted but with my highlights:
TruckerGuy:
Data from the study of SARS-CoV and other respiratory viruses suggest that anti-SARS-CoV-2 antibodies could exacerbate COVID-19 through antibody-dependent enhancement (ADE). Previous respiratory syncytial virus and dengue virus vaccine studies revealed human clinical safety risks related to ADE, resulting in failed vaccine trials.
…
ADE has been observed in SARS, MERS and other human respiratory virus infections including RSV and measles, which suggests a real risk of ADE for SARS-CoV-2 vaccines and antibody-based interventions. However, clinical data has not yet fully established a role for ADE in human COVID-19 pathology.
So agrees there is no proof of ADE in C19…although we are still looking…and if a vaccine is risky it fails the trials…
Well? The system works. Good job too!
This post is getting long. Coffee time.
that’s way I don’t come on here much now 